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Recent Posts
- Science: “Volatile Chemical Products Emerging as Largest Petrochemical Source of Urban Organic Emission,” McDonald et. al., February 16, 2018
- EurekAlert!: “Consumer and Industrial Products Now a Dominant Urban Air Pollution Source,” University of Colorado at Boulder, February 15, 2018
- Science Daily: “Air Pollution May Shorten Telomeres in Newborns — A Sign of Increased Health Risks,” Columbia University Mailman School of Public Health, January 24, 2018
- NEJM: “Air Pollution and Mortality in the Medicare Population,” Di et. al., June 29, 2017
- Stroke: “Long-Term Exposure to Fine Particulate Matter, Residential Proximity to Major Roads and Measures of Brain Structure,” Wilker et. al., April 23, 2015
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- Particulate Pollution and Lactic Acid Dehydrogenase
- Particulate Pollution and Lung Cancer
- Particulate Pollution and Lung Function
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- Particulate Pollution and Mortality
- Particulate Pollution and Multiple Chemical Sensitivity
- Particulate Pollution and Multiple Sclerosis
- Particulate Pollution and Myocardial Infarction
- Particulate Pollution and Nasal Pathology
- Particulate Pollution and Nervous System
- Particulate Pollution and Neurodegeneration
- Particulate Pollution and Neurogenic Inflammation
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- Particulate Pollution and Omega-3 Fatty Acids
- Particulate Pollution and Oxidative Stress
- Particulate Pollution and Parkinson's Disease
- Particulate Pollution and Pneumonia
- Particulate Pollution and Pregnancy
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- Particulate Pollution and the Nervous System
- Particulate Pollution and Transient receptor potential vanilloid-1 (TRPV1)
- Particulate Pollution and Triglycerides
- Particulate Pollution and TRPA1
- Particulate Pollution in China
- Particulate Pollution No Safe Level
- Phthalate Ester-Associated Asthma
- PM2.5 and Brain Volume
- PM2.5 and Chronic Kidney Disease
- PM2.5 and eGFR Decline
- PM2.5 and End-Stage Renal Disease
- PM2.5 and Kidney Disease
- PM2.5 and Mortality
- PM2.5 and Stroke
- PM2.5 No Safe Level
- Polycyclic Aromatic Hydrocarbons and Child Behavior
- Polycyclic Aromatic Hydrocarbons and Disease
- Polycyclic Aromatic Hydrocarbons and Intelligence
- Pterostilbene and asthma
- Residential Wood Smoke Pollution
- Respiratory Protection and Woodsmoke
- Rheumatoid Arthritis and COPD
- Second Hand Smoke and Risk of Multiple Sclerosis
- Serious Non-Attainment
- Smoke Pollution and Americans with Disabilities Act
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- Smoking and Eye Disease
- Solid Fuel Use and Cardivascular Disease
- State of Alaska Proposed Air Quality Regulations
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- Stuart Creek Fire
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- Substance P and Asthma
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- Temperature Inversions and Air Quality
- The Secret Life of a Soot Particle Video The Allegheny Front
- Tobacco Smoking and Death Rate
- TRP Channels and Environmental Stimuli
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- Uncategorized
- Utah Physicians for a Healthy Environment
- Vitamin D and Air Pollution
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- VOCs and Air Pollution
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- Wickersham's Conscience and Fairbanks AIr Pollution
- Wildfire Health Risks
- Women and COPD
- Wood and Coal Smoke and Lung Cancer
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- Wood Smoke and Mortality
- Wood Smoke and Neurodevelopment Performance
- Wood Smoke and Pneumonia
- Wood Smoke and Respiratory Infection
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- Wood Smoke and Wildland Firefighters
- Wood Smoke Carcinogens
- Wood Smoke Costs
- wood smoke deposition fraction
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- Woodsmoke and Children
- World Health Organization
Meta
Tag Archives: early markers of neurodegenerative disease
Air Pollution and the Brain: Subchronic Diesel Exhaust Exposure Causes Neuroinflammation and Elevates Early Markers of Neurodegenerative Disease
From: Journal of Neuroinflammation, 2011. http://www.jneuroinflammation.com/content/8/1/105 Journal of Neuroinflammation 2011, 8:105 doi:10.1186/1742-2094-8-105 Air Pollution & the Brain: Subchronic Diesel Exhaust Exposure Causes Neuroinflammation and Elevates Early Markers of Neurodegenerative Disease Shannon Levesque1, Michael J Surace1, Jacob McDonald2 and Michelle L Block … Continue reading
Posted in Particulate Pollution and Brain Damage, Particulate Pollution and Inflammation
Tagged cytokine, early markers of neurodegenerative disease, glial cell, inflammation, midbrain, neuroinflammation, particulate air pollution, subchronic diesel exhaust exposure, systemic inflammation, tumor necrosis factor alpha
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